Dr. Peter Simson
1) Neuropsychopharmacology of ethanol action, 2) Biological bases of affective disorders (in particular, changes in brain electrophysiology associated with depression), and 3) Noradrenergic modulation of memory.
Schneider, A.M., Simson, P.E., Atapattu, R. and Kirby, L. (2011) Stress-dependent modulation of passive-avoidance memory by propranolol or naloxone. Pharmacology, Biochemistry and Behavior, 98, 539-543.
Schneider, A.M., Simson, P.E., Spiller, K., Adelstein, J., Vacharat, A., Short, K., and Kirby, L. (2009) Stress-dependent enhancement and impairment of retention by naloxone: evidence for an endogenous opioid system protective of memory. Behavioral Brain Research, 205, 290-293.
Schneider, A.M., and Simson, P.E. (2007) NAN-190 potentiates the impairment of retention produced by swim stress. Pharmacology, Biochemistry and Behavior, 87; 73-80.
Schneidr, A. M., & Simson, P. E. (2000). beta-noradrenergic blockade with propranolol enhances retention in a multi-trial passive-avoidance memory paradigm. Behavioral Neuroscience.
Matthews, D. B., & Simson, P. E. (1998). Prenatal exposure to ethanol disrupts spatial memory: effect of the training-testing delay period. Physiology and Behavior, 64, 63-67.
Yang, X., Criswell, H. E., Simson, P. E., Moy, S., & Breese, Gr. R. (1996). Evidence for a selective effect of ethanol on NMDA responses: ethanol affects a subtype of the ifenprodil-sensitive NMDA receptor, Journal of Pharmacology and Experimental Therapeutics, 278, 114-124.
Matthews, D. B., Simson, P. E., & Best, P. J. (1996). Ethanol alters spatial processing of hippocampal place cells: a mechanism for impaired navigation when intoxicated. Alcoholism: Clinical and Experimental Research, 20, 404-407.